Lowprotein

non all naturally occurring metals are used by the personate for important biological processes. Lead and compact disk are examples of these metals that are not essential for life but may even cause toxicity and death if taken in large doses. A study conducted in Japan showed a coefficient of correlation between protein intake with the increased vulnerability for intoxication of candle (Tavari 1986). Rats given a low protein forage were observed to have higher toxicity from these metals that were also detected in the urine and feces (Suzuki 1984). A low protein diet in humans is usually advised to those with kidney and liver diseases and as a result, this prejudicial effect of higher metal toxicity is important to understand.Metals act by binding to organic compounds afterward altering their structure and by chance modifying their function. When the function in not carried emerge well, this can lead to cubicle death and inactivation of the production of important enzymes (Metals as toxins). For instance, a metal compound can cope with a biologically significant element such as oxygen to create an enzyme responsible for contaminating glucose. If this metal successfully defeats oxygen, the enzyme may not be produced thus, glucose will not be degraded and possibly accumulated. This is a simple example of what a metal can do to the body.In the case of regulation protein intake, the body has enough proteins that can bind to defamatory compounds such as metals. Similar to the action of a lock and key, a specific protein can complex with damaging bioelements and then excrete them outside the body to prevent possible internal damage. Metallothionein specifically works as a chelating agent and combines itself with cadmium, for example and is excreted out of the body while Selenium, a protein abundantly found in egg whites, suppresses the toxic effect of metals (Metals as toxins). A low protein intake thus, have a significant effect in resulting to high l evels of cadmium and lead in the blood since most protein clearly function in sweeping out these harmful metals by binding with them and then carrying them out of the body.ReferencesMetals as toxins. Retrieved stately 11, 2007, fromhttp//www.portfolio.mvm.ed.ac.uk/studentwebs/session2/group29/introtox.htmSuzuki, K.T., Miyamoto, E., Tanaka, Y. Kawamura, R. and Yamamura, M. (1984). Effect of dieton urinary and fecal excretion of cadmium, copper and zinc from rats preaccumulatedheavily with cadmium. Archives of Environmental befoulment and Toxicology, vol.13 no.5. Retrieved August 11, 2007, fromwww.springerlink.com/index/J456157JVU87212T.pdfTavari, P.C., Jain, V.K., Ashquin, M. and Tandon, S.K. (1986). Influence of protein deficiencyon cadmium toxicity in rats. Archives of Environmental Contamination and Toxicology,vol.15 no.4. Retrieved August 11, 2007, fromwww.springerlink.com/index/G88L4P84417XT467.pdf